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The Low-Down on Inflammation from an HIV Doctor

Inflammation is the generic term for the body’s response to injury. During injury, the immune system—our body’s defense system—activates a complicated network of cells and chemical signals. Acute inflammation, immune activation that’s rapid and self-limited, is essential for healing. But chronic inflammation, immune activation that continues even after the initial injury is gone, is problematic. Chronic inflammation is like a volume control knob on a stereo being stuck—with the volume turned all the way up.

Published
24 August 2015
From
BETA blog
Targets proliferate in HIV cure research

The Towards an HIV Cure two-day symposium has become a fixture in advance of the International AIDS Society conferences and this one featured a more varied range

Published
21 July 2015
By
Gus Cairns
Young woman stays undetectable for twelve years off treatment after early HIV therapy

The 8th International AIDS Society Conference on HIV Pathogenesis, Treatment and Prevention (IAS 2015) heard today of a case where a young woman, who was infected with

Published
21 July 2015
By
Gus Cairns
New drug holds out promise of long-term control or even cure of HIV

Scientists at the Scripps Research Institute in California have discovered a new type of drug that may permanently inhibit HIV from becoming reactivated in the cells that

Published
18 July 2015
By
Gus Cairns
HIV uses the immune system’s own tools to suppress it

A research team at the IRCM, led by molecular virologist Éric A. Cohen, PhD, made a significant discovery on how HIV escapes the body’s antiviral responses. The team uncovered how an HIV viral protein known as Vpu tricks the immune system by using its own regulatory process to evade the host’s first line of defence. The findings, published yesterday in the scientific journal PLOS Pathogens, pave the way for future HIV prevention or cure strategies.

Published
16 July 2015
From
Institut de Recherches Cliniques de Montreal
Potential New HIV Therapy Seen in Component of Immune Cells

A research team led by Weill Cornell Medical College scientists has discovered a way to limit replication of the most common form of HIV at a key moment when the infection is just starting to develop. The study, published June 25 in Nature Communications, has shed light on a potential new element of human immunity against HIV-1 and could provide a powerful new strategy — perhaps as part of an HIV vaccine — to limit the severity of the disease.

Published
26 June 2015
From
Weill Cornell Medical College
Life as an Elite HIV Controller

After finding out that I was an elite controller, I felt a lot of confusion. I knew what an elite controller was, but that didn’t translate to knowing what it actually meant to be one. I wasn’t new to the HIV world—I had previously been involved in HIV research—but I felt like I knew nothing about what was going on in my own body.

Published
24 June 2015
From
BETA blog
Changes in HIV genetic code determine severity of disease

In a finding that furthers the understanding of human immunodeficiency virus (HIV), researchers from Children's Hospital Los Angeles discovered two locations where a single difference in HIV's genetic code altered the way the virus infected the cell, thereby influencing the progression of the disease.

Published
18 June 2015
From
Eurekalert Inf Dis
Dendritic cells of elite controllers able to recognize, mount defense against HIV

Investigators from Massachusetts General Hospital and the Ragon Institute of MGH, MIT and Harvard have added another piece to the puzzle of how a small group of individuals known as elite controllers are able to control HIV infection without drug treatment. The research team reports finding that dendritic cells of elite controllers are better able to detect the presence of HIV, which enables them to stimulate the generation of T cells specifically targeting the virus.

Published
12 June 2015
From
Eurekalert Inf Dis
HIV can spread early, evolve in patients' brains

HIV can genetically evolve and independently replicate in patients' brains early in the illness process, an analysis of cerebral spinal fluid has found. Prompt diagnosis and treatment with antiretroviral therapy should reduce the risk that the virus could find refuge and cause damage in the brain, where some medications are less effective -- potentially enabling it to re-emerge, even after it is suppressed in the periphery, say researchers.

Published
27 March 2015
From
Eurekalert Inf Dis
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